Sentence examples for cancer selection from inspiring English sources

In (47) Is Graham really claiming that cancer selection is the only driving force in the emergence of complex animal life?

Issues of importance in these smaller trials include choice of agent, selection of appropriate trial participants, trial design, method of access to breast tissue in women without cancer, selection and monitoring of SEBs, and monitoring of drug toxicity.

In cancer, selection at the level of the cell type differs from selection at the level of the host organism because the role of the affected cell type in the multicellular organism is at odds with maximizing the number of its progeny cells.

Except for luminal A breast cancer, selection of essential medicines did not allow treatment of early breast cancer subtypes in many LMICs.

There are of course a number of competing selective pressures acting on human populations (as well as a bottle neck in population size that can contribute to fixation of slightly deleterious mutations), and while pathogen load has been proposed as the most dominant driver of adaptation in the human lineage [ 70], cancer selection has a distinct possibility as a contributing factor [ 69, 73].

As during progression of cancers selection operates favouring cells with activated oncogenes, it is not surprising that cells showing the activation of a HER2 downstream signal, such as mTOR, do not require the simultaneous activation of the upstream signal, i.e. HER2.

Altogether, these examples suggest that there might be a direct or indirect relationship between cancer and selection, but the overall genome-wide proportion of cancer-related genes actually targeted by selection remains to be determined.

Although the response rate was 69.7% for breast cancer cases, selection (inclusion) of cases was not related to exposure status (i.e. antioxidant intake), therefore, selection bias is unlikely to occur.

Unfortunately, in prostate cancer, such selection has been a difficult-to-impossible task to date.

The current perspective considers cancer as selection biased differential fitness between heterogeneous populations of clonal lineages that arises as a function of genetic mutation and epigenetic alterations [3].

Aim: Detection of the extent of local recurrence and of metastases in biochemical recurrence (BCR) of prostate cancer facilitates selection of appropriate treatment.