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Mitochondrial calcium overload plays an important role in the oxidative stress-induced neuronal death.
MDA-related carbonyl stress injures neurons by triggering Ca2+ influx and calcium overload [39].
It is now generally assumed that this calcium overload produces the ventricular dysfunction in catecholamine cardiotoxicity.
Intracellular myocytes calcium overload due to catecholamine enhancement has been observed in myocardial biopsy specimens [30].
Our findings provide a new perspective in understanding calcium overload in myocardial ischemia/reperfusion.
This stunned myocardium could be the consequence either of an intramyocardial calcium overload or ischemic-reperfusion phenomena [12 14].
Catecholamines can decrease the viability of cardiomyocytes through cyclic AMP-mediated calcium overload and oxygen-derived free radicals [14].
We suggest that the presence of CR alone is not sufficient to spare neurons from a toxic calcium overload.
Calcium overload and oxidative insult are the two mutually non-exclusive phenomena suggested to cause cardiac dysfunction.
It's almost a calcium overload.
The Dr. Oz Show Pancreatic cancer; calcium overload; protein powder.
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