Sentence examples for cad number from inspiring English sources

For example, Simova and colleagues [ 33] found only a weak correlation between functional and morphological parameters and this was no longer significant when adjusting for presence of CAD, number of diseased coronary arteries and percentage coronary artery stenosis [ 33].

There was a significant linear trend of ECI, and ECI ≥7 has in MDCT a greater presence of both severe calcified wall and obstructive CAD, number of affected vessels, and mixed/calcified plaques (all P < 0.001).

The CCORP data collection system includes questions on the presence or absence of left main CAD, number of diseased coronary vessels, left ventricular ejection fraction (LVEF) and Canadian Cardiovascular Society classification of angina severity.

The prognostic value of the Killip classification is highlighted in terms of the extent of CAD (number of affected vessels and revascularization technique), of NYHA class and, last but not least, of HRQoL.

The frequency distributions of NGT, IGT and T2DM among patients grouped by the presence of CAD and number of stenosed coronary arteries were illustrated.

Coronary angiography was performed to assess the extent of CAD (the number of lesions in the coronary arteries with more than 50% stenosis as well as the patency of the infarct-related artery according to TIMI flow grading system).

However, as in the case of CAD, a number of investigations addressing the role of the HNF1a gene polymorphism with respect to diabetic disease conditions have yielded somewhat inconsistent results.

Among the identified secretome-specific spots, there was the sE-cad (accession number: gi|6682963; experimental versus theoretical pI and MW: 4.2/4.6 and >85/99.7).

The investigators found that MIP and CAD reduced the number of overlooked nodules compared with LDCT and that the MIP was more sensitive than the CAD.

Twenty-four hours after ROSC, the mean numbers of EMPs (69.1 ± 12.4 events/μL) in resuscitated patients were significantly higher than in the CAD group (mean number 22.0 ± 3.0 events/μL; P < 0.005) and in healthy subjects 15.4 ± 4.4 events/μL; P < 0.001; Figure 5).

A similar strategy was used in the present study, suggesting that in subjects with advanced CAD, an increasing number of prothrombotic alleles may confer a significant risk of developing MI.