We examined the association of inflammation and progression of coronary artery calcification (CAC)—a marker of subclinical atherosclerosis in adults with and without type 1 diabetes.
Primers 5'- CAC GAG TGG GTT ACA TCG AAC TGG -3' and 5'- CAA TAC GGG ATA ATA CCG CGC CAC -3' (A primers) flanked a control region on the plasmid (see Fig. 1B).
The Community Action Cycle (CAC), a comprehensive strategy, empowers communities to take charge of problems within their own context.
We hypothesized that the Hp genotype would predict progression of coronary artery calcification (CAC), a marker of subclinical atherosclerosis.
Specifically, we measured CAC a median of 10 years after circulating vitamin D metabolites and thus tested associations of vitamin D metabolites with a combination of prevalent and incident CAC in a staggered cross-sectional analysis.
Lower β-cell function is associated with microalbuminuria, a microvascular complication, while impaired insulin sensitivity is associated with higher CAC, a predictor of macrovascular complications.
In contrast, CACTI has used CAC, a marker of subclinical coronary atherosclerosis shown to predict cardiovascular outcomes, as a surrogate marker of CVD (23, 24).
Coronary artery calcification (CAC), a marker of coronary artery plaque burden, predicts coronary events in persons with type 1 diabetes (3).
Coronary artery calcium (CAC), a marker of atherosclerosis, has been shown to independently predict cardiovascular events as well as enhance risk stratification in patients with diabetes (2– 5).
To investigate the requirement for inflammation in the susceptibility of Gpa33 −/− mice to CAC, a model of sporadic CRC was employed.
