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Circulating adiponectin levels are positively correlated with insulin sensitivity evaluated by using different insulin sensitivity techniques (28).

In one study (12), subjects randomized to adjustable gastric banding plus omentectomy had a greater improvement in oral glucose tolerance and insulin sensitivity, assessed by using an intravenous insulin tolerance test, than subjects randomized to adjustable gastric banding alone.

Insulin sensitivity was additionally estimated from OGTT data by using the oral glucose insulin sensitivity index (OGIS) [ 25] and insulin secretion from OGTT data was assessed by the insulinogenic index (IGI = Δinsulin30/Δglucose30) [ 26].

Indirect measures of insulin resistance and insulin sensitivity were calculated by using homeostasis model assessment (HOMA) and quantitative insulin sensitivity check index (QUICKI), respectively.

Insulin resistance was assessed by using Homeostasis Model Assessment of Insulin Resistance (HOMA-IR) and Quantitative Insulin Sensitivity Check Index (QUICKI) in subjects without a history of diabetes before inclusion into the study.

Indices of β-cell function, insulin sensitivity (SI), and insulin clearance rates were estimated by using minimal models of glucose, insulin, and C-peptide kinetics.

We also did not differentiate between hepatic and peripheral insulin sensitivity by using stable isotopes in the clamp studies.

Insulin sensitivity was measured using the insulin sensitivity index of Matsuda, a well-established measure of whole-body insulin sensitivity [ 16].

Insulin sensitivity was calculated using the insulin sensitivity index composite (ISIcomp: 10.000/√ G0 ⋅ I0 ⋅ Gmean ⋅ Imean).

Insulin sensitivity was measured using the insulin sensitivity index (ISOGTT) of Matsuda and DeFronzo (10, 11).

Insulin sensitivity was measured using the insulin sensitivity index (ISOGTT) of Matsuda and DeFronzo (11).