With stratified analyses by three age categories, associations of largest magnitude were observed in the youngest age group (20 50 years old) in both symptomatic OA (adjusted OR=10.36, p<0.001) and OA-related surgery (adjusted OR=6.20, p=0.007) (table 3).
In the Cancer Prevention Study II, stratified analyses by three age groups (30 64 years, 65 74 years, and ≥75 years) showed stronger association of mortality associated with increasing BMI in younger than older participants (Calle et al., 1999).
Tables 5 and 6 repeat the same analyses presented in Tables 2 and 4, respectively, but stratified by two age groups, ages 0 4.9 and 5 14.9.
To reduce the potential for age-related confounding, we also performed stratified analyses in three age strata defined by 20-year age ranges (20 39, 40 59, and ≥60 years).
For the four age subgroup analyses, all pooled results are from random-effects models.
Other fields included in analyses included age by ten year age group; sex; type of diabetes including Type 1, Type 2 and 'Other and not specified'; duration of diabetes by five year group and a category for not known.
We present here one of the first comprehensive proteomic analyses of the hippocampal region of three brains affected by AD and three age-matched controls.
We did subgroup analyses by stratifying the entire cohort into two age groups (<65 and ≥65 years).
We stratified mortality analyses by sex and seven age groups (neonates, infants, and ages 1 4, 5 14, 15 49, 50 64 and over 65 years).
Analyses were centre and age adjusted by including each of these covariates in regression models as four dummy variables, distinguishing the five study centres and five age categories.
If all cell values in the 2×2 table were at least five, we stratified analyses by age 40-699 and ≥70).
