Sentence examples for by these cells which from inspiring English sources

CB2 receptor activation augments the production of the anti-inflammatory cytokine, IL-10, by murine macrophages [22], and disrupts antigen processing by these cells, which leads to incomplete antigen-presentation to T cells [23], [24].

One hallmark of anti-tumor cell infiltrates is increased nitric oxide (NO) production by these cells, which enhances tumoricidal activity, while pro-tumor infiltrates are poor producers of NO [44], [45], [46].

In light of these studies and our present data, we propose that, in vivo, allergen exposure in sensitized asthmatics not only enhances neutrophil recruitment to the site of allergen exposure but also upregulates expression of FcεRI by these cells which may therefore participate in IgE-mediated allergic inflammatory responses.

Combinations of IL23 and IL1α induced production of cytokines by these cells, which increased further after administration of IL6.

We demonstrated significantly reduced expression of cytokine mRNA by these cells, which was paralleled by decreased levels of at least some proinflammatory cytokines in BAL fluid.

This was done by adding serum into the culture medium and by further passaging these cells, which were initially maintained in a serum-free condition [ 3, 7].

VEGFR-1 is also expressed by monocytes/macrophages and osteoclast precursor cells [ 40] and it is possible that angiogenesis and tissue damage in the rheumatoid joint is largely mediated by infiltration with these cells which then secrete further angiogenic and catabolic factors, rather than by direct interaction with VEGFR-2 on endothelial cells.

However, only the full-length protein was detected by Western blotting in these cells, which does not support the hypothesis that ICC detects a truncated cytoplasmic protein.

In particular, we found that two internalization events anterior to the ABprp cells were accompanied by the anterior movement of these cells, which was about 5% of the embryo length.

hTERT was expressed at similar levels in both EP156T and EPT1 and the exogenous nature was supported by very high puromycin resistance of these cells, which is consistent with hTERT expression from a puromycin resistance retroviral vector (data not shown) [20].

This result was further confirmed by the transfection of ERα into these cells which restored their response to CLA.