Highly porous scaffolds have been fabricated by the replication technique using 45S5 Bioglass® (BG) powder.
During PARP inhibition, DNA lesions that induce fork arrest and are normally resolved or repaired remain unrecognized by the replication machinery.
In contrast, the heart, liver, and pancreas increase their cellular mass in part by the replication of differentiated cells that also perform the organ's function3,4,5.
We also present a rare object search algorithm to reduce the overhead caused by the replication strategy.
Three-dimensional (3D), highly porous, mechanically competent, bioactive and biodegradable scaffolds have been fabricated for the first time by the replication technique using 45S5 Bioglass® powder.
Mde2, whose expression is strictly regulated by the replication checkpoint, interacts with Rec15 to stabilize the SFT subcomplex and further binds Rec14 in DSBC.
a, During damage tolerance, damaged sites are recognized by the replication machinery before they can be repaired, resulting in an arrest that can be relieved by replicative bypass (translesion DNA synthesis) (see Fig. 3).
The validity of our metabolomics pipeline is supported by the replication of reported OGTT metabolite trajectories including the overall decline of acylcarnitine5,6,23 and the expected association between IR and hexose.
This novel phenomenon is interpreted in terms of drastic change in dislocation mobility and rearrangement, which is supported by the replication for slip bands and TEM observations for dislocation structure.
Recruitment of the xFA core complex and xFANCD2 to chromatin is strictly dependent on replication initiation, even in the presence of MMC indicating specific recruitment to DNA lesions encountered by the replication machinery.
Here we describe a group of host factors designated as 'NFAR proteins' that are recruited by the replication machinery of bovine viral diarrhea virus, a close relative of the human pathogen hepatitis C virus.
