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Genes regulated by Mta1 under different conditions were identified by performing gene expression analysis followed by the Gene Ontology analysis.
By performing gene expression profiling on these cells we are able for the first time to ask what genes and pathways are 'turned on' in these cells, and these may be key pathways that are required for them to migrate out of the tumor in the first place.
As we observed that the refined depletion protocol affected such a basic function of IECs as the regenerative activity, we wanted to study how the intestinal microbiota affected IECs globally by performing gene expression profile analysis on isolated colonic IECs of mice successfully depleted by the refined microbiota depletion protocol compared with that of sham treated control mice.
This might be possible by performing gene expression profiling using QRT PCR or microarray analyses.
In this study, we identified several genes that display DMH-enriched expression patterns by performing gene expression profiling with RNA samples collected from laser-microdissected hypothalamic nuclei.
We therefore chose an immature, stem-like mesenchymal cell line with adipogenic potential to investigate whether the wild type and truncated forms of the protein activate the same pathways, by performing gene expression analysis.
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To identify the genes whose estrogen regulation in hormone-responsive BC cells is perturbed by ERβ, we performed gene expression profiling with microarrays in estrogen-starved, quiescent wt and TAP-ERβ+ (Ct-ERβ and Nt-ERβ) MCF-7 cells following stimulation with 10-8M E2.
In addition, to better understand molecular mechanism of FAK down-regulation by FAKsiRNA, we performed gene expression of MCF-7, MCF-7-Vector, MCF-7-Control siRNA, FAKsiRNA#1 and FAKsiRNA#2 cell line samples by microarray analysis using Affymetrix Human Genome U133 GeneChip Array, covering the whole human genome with >47000 transcripts.
To elucidate potential molecular networks regulated by HDACs, we performed gene expression profiling of the differentiating cells treated with MGCD on day 3 and day 7 (D3M and D7M), as well as the control cells at the same time points of neural differentiation (D3C and D7C).
We believe 3SEQ could revolutionize the field of translational cancer genomics, by allowing investigators to perform gene expression profiling on large numbers of well-annotated archival tumor specimens with long term follow-up.
In order to identify genes and pathways affected by maternal diabetes, we performed gene expression profiling of diabetes-exposed mouse embryos using oligonucleotide microarrays.
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