Because TP03_0871 is identical to TP03_0884, the transcript detected by microarray analysis suggests that one or both genes are expressed.
However, somewhat unexpectedly, up-regulation of phytochelatin synthase was not detected by microarray analysis suggesting that PCs are not involved in Cr (VI) detoxification.
These findings lead us to conclude that tumors 5A and 5B are essentially indistinguishable by microarray analysis, suggesting that they are likely to be derived from the same clone.
Altered transcription of components of the Akt/GSK3-β regulatory pathway was consistently observed in flies expressing rCAG, rCUG and rAUUCU repeat RNAs by microarray analysis, suggesting that this is a key component of cellular dysfunction in this Drosophila model of untranslated repeat disease pathogenesis.
The genetic repertoire of the FGI indicated by the microarray analysis suggested that the glycan biosynthetic capacity of these C. botulinum strains may vary.
Second, no col genes with peak expression in the embryo, L1, L2, or dauer larva were found as BAR-1 responsive genes by the microarray analysis, suggesting these genes are not sensitive to ectopic Wnt signaling at the L2/L3 molt (Table S1).
Therefore the combined induction of GADD45α, GADD45β and GADD153 by Onconase seen in our microarray analysis suggests these genes may act synergistically to inhibit MM cell viability.
Although spalt and E75 were the only two transcription factor genes for which we were able to detect marking-associated expression by whole-mount in situ hybridization, microarray analysis suggested that E spl) region transcript mbeta and fringe, both involved in the Notch signaling pathway, also have marking specificity.
Microarray analysis suggests that the processes engaged by this sleep are consistent with its function.
The results of the microarray analysis suggested that both lmo0302 and lmo0303 are negatively affected by LhrA (2.5 fold and 2.2 fold, respectively, see Table S2).
