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Gene expression changes detected by microarray analysis revealed induction of FGFR2 and FGFR3 but not FGFR1, FGFR4, or FGFR ligands in gefitinib treated cells (Table S1).
Analysis of gene regulation by microarray analysis revealed a large number of genes that were both up- and down-regulated (>3-fold) after HIF-1α-PPN expression for either 1 day or 3 days (GEO reference GPL7119).
Even the re-annotation of the contigs identified by microarray analysis revealed a total of 41% novel proteins.
Comparative gene expression studies by microarray analysis revealed that 19% of the genes analyzed in wheat showed more than a 5 fold difference in expression between homeologous gene copies [ 24].
Furthermore, our ChIP-chip analysis (chromatin immunoprecipitation followed by microarray analysis) revealed that Hes1 directly binds to Dll1 and Jag1 promoter regions in ES cells but not to Notch1 or Hes5 promoter regions (Kobayashi et al. 2009).
Accordingly, genome comparisons by microarray analysis revealed that genome content of clade VI strains is more similar to rodent and pig strains, reflecting similarities in lifestyle, probably related to similarities in biofilm formation in the proximal GI tract of these animals.
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Six imprinted genes were among the forty most differentially expressed transcripts as determined by microarray analysis, revealing the relative efficiency of such an approach in identifying novel imprinted genes.
The first insights into the intracellular life of M. tuberculosis provided by comparative microarray analysis, revealed a switch from aerobic to anaerobic respiration, induction of the dormancy regulon dosR and iron scavenging as well as upregulation of β-oxidation of fatty acids upon infection [ 5].
After controlling false discovery rate by Benjamini-Hochberg correction (p < 0.05), microarray analysis revealed 528 genes were up-regulated and 15 genes were down-regulated in MUC5AC high expressors compared to the MUC5AC low expressors.
Microarray analysis revealed many novel genes differentially expressed by ventilation including matrix metalloproteinase-8 (MMP8) and GADD45α.
Our microarray analysis revealed activation of mostly identical genes by IL-28A and IL-29.
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