Sentence examples for by interacting with complementary from inspiring English sources
Despite being small (~22 nt) microRNAs (miRNAs) profoundly influence tissue-specific gene expression by interacting with complementary target sequences in cellular messenger RNAs, impairing their translation or marking them for early destruction.
Here, we show that ncRNAs can regulate gene expression in the hyperthermophile Sso by interacting with complementary sequences present in the 3′ UTR of ORF 1183.
Recent data show that miR regulation entails far more complex posttranscriptional control, with the ability to both repress and activate gene expression by interacting with complementary sequences in coding and noncoding regions of their mRNA targets [ 115].
It has been found that miRNAs regulate the expression of target genes by interacting with complementary sites in the 3'-untranslated region (UTR) of target mRNAs [ 5], and more than 30%% of human genes are regulated post transcriptionally by miRNAs [ 6]. miRNAs may function as oncogenes or tumor suppressors by targeting many cancer-associated genes in the progression of EHCC.
MicroRNAs are an important class of regulatory RNAs which repress animal genes by preferentially interacting with complementary sequence motifs in the 3' untranslated region (UTR) of target mRNAs.
MicroRNA are an important class of regulatory RNA that repress animal genes by preferentially interacting with complementary sequence motifs in the 3' UTR of target mRNA [ 58].
Besides, EIciRNAs (exon-intron circRNAs) have analogous effects to parent gene transcription by interacting with U1 snRNP and Pol II through complementary sequence pairing between EIciRNA and U1 snRNA (Fig. 1, function I) (Li et al., 2015).
They exert their effects by interacting with target sequences in mRNA 3′ UTRs (untranslated regions) via a complementary 6 8 bp 5′ 'seed region' and variable imperfect complementarity in the rest of the ~22 nucleotide sequence [ 1].
An additional, perhaps complementary, pathway may also exist by which P influences anxiety by interacting with PR.
In this model, an RNA molecule, complementary to the rRNA promoter mediates de novo CpG methylation of rRNA genes by interacting with the transcription factor TTF-1 and forming a DNA RNA triplex, which recruits the DNA methyltransferase DNMT3b.
