Sentence examples for by increasing cell viability from inspiring English sources

Knock-down of SSTR1 in gastric cancer cell lines significantly induced cell growth by increasing cell viability and clonogenicity.

We found that pretreatment with RSV suppressed tert-butyl hydroperoxide (t-BHP -induced oxidat-BHP -induced human umbilical vein endot-BHP -induced(HUVECs) by increasing cell viability, inhibiting cell apoxidativerepressing collapse of mitochondamagemembrane potentinl and decreasing mtROS generation.

Incubation with geniposide showed a dose-dependent increase in cell viability, with increasing cell viability by 22% at 100  μM measured by CCK-8 assay.

Pre-treatment with zVad significantly rescued both cell lines from TRAIL/bortezomib-induced cell death, increasing cell viability by ~30%.

Antisense knockdown of miR-466h-5p miR-466h-5p miR-466h-5p in nutrient-delayedd conditions by decreapoptosispase-3/7 activationsetd increasing cell viability (Druz et al., 2011).

The BMP-2/HAp TiO2 BMP-2/HAp TiO2outperforms systems of pure Ti, BMP-2/Ti, and hybridO2 by promoting cell adhesystemncreasing filoutperformspolygonal lamellipodia extensystemsnd increasing cell viability.

In contrast, knockdown of hCLP46 by siRNA increased cell viability and almost totally blocked the inhibition of cell proliferation by TGF-β1 (Fig. 2B).

Our results in the present work showed that EEP remarkably restrained ox-LDL-induced lipid cumulation and macrophage insult as reflected by the increased cell viability and the decreased LDH leakage, caspase-3 activation and apoptosis, suggesting that EEP is able to attenuate ox-LDL-induced macrophage-derived foam cell injury.

However, JNK phosphorylation inhibition by SP600125 supplementation to pM treatment remarkably increased cell viability by blocking pM-induced cell death in both 4910 (from pM; 36.6±2.8 to pM+SP600125; 61.5±1.5%) and 5310 (from pM; 35.5±2.0 to pM+SP600125; 62.6±3.3%) cells (p<0.05).

Since glutathione is a powerful intracellular antioxidant [ 45], it was expected that if the IFNQ was working via a redox mechanism then its effects would be antagonized by glutathione resulting in increased cell viability.

Inhibition of ascorbate-induced autophagy by 3-MA treatment increased cell viability, and knockdown of Bif-1, a positive mediator of autophagy, resulted in PC-3 cell resistance to ascorbate-induced cell death, thus suggesting a prodeath role for autophagy [ 52].