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To examine whether bronchial hyperresponsiveness (BHR) in patients with chronic fatigue syndrome (CFS) is caused by immune system abnormalities.
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Systemic lupus erythematosus (SLE) is an autoimmune disease characterized by multiple immune system abnormalities, including production of autoantibodies that can lead to inflammation and tissue damage [ 1].
Nevertheless, research into immune system abnormalities in Rett syndrome has been surprisingly limited.
The pathogenesis of BD remains unknown but major determinants involving genetic and immune system abnormalities have been recently reported [ 20].
Within recent years, search for innate immune system abnormalities in rheumatoid arthritis (RA) has attracted considerable attention [ 1].
Head and neck squamous cell carcinoma (HNSCC) is an immunosuppressive malignancy characterized by tumor-driven immune-system abnormalities that contribute to disease progression.
Autoinflammatory diseases are characterised by hyperactivation of the innate immune system and abnormalities in pro-inflammatory cytokine signalling.
Numerous disorders in the immune system and abnormalities in cytokine productions have been described in patients with SLE.
Systemic sclerosis (SSc) is a multisystem disease characterized by vascular abnormalities, immune system activation manifested by SSc-specific autoantibodies and disturbances in fibroblast function.
Allergies are overreactions by the immune system against organic substances.
How is chitin recognized by the human immune system?
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com