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Thus, we explored their potential role in OSCC by evaluating their expression levels in several OSCC cell lines compared with normal human oral keratinocytes.
The cells were confirmed as MSCs by evaluating their expression marker (CD 105, CD 73, HLA-DR, CD 34, CD 45, CD 14, and CD 19).
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We evaluated their expression by immunohistochemistry and found that ELF-EMF altered the amounts of positive cells expressing these proteins and we concluded that this effect is associated with the diminishing of cell proliferation.
We evaluated their expression levels by quantitative RT-PCR and found that they were both significantly elevated in OA chondrocytes compared with normal chondrocytes (P < 0.05).
Possible mechanism(s) responsible for the antiproliferative, pro-apoptotic, and cell cycle regulatory effects of the four compounds were investigated by evaluating their effects on the expression of various proteins involved in these processes.
In the present study, we investigated the potential regulatory functions of NK cells in the Th1-biased environment of BD by evaluating their activation status, gene expression profiles, and functional properties in association with the disease status.
By evaluating their surface marker pattern and expression of iNOS and TNF we could proof that the IFNβ producing cells were a subpopulation of Tip-DCs with T cell priming abilities of professional APCs.
To determine whether the defects in NK cell maturation and function were associated with a reduction in expression of these key transcription factors, we evaluated their intracellular expression by flow cytometry.
To validate the results of the in silico expression analysis, we selected ten transcripts in the PERNI data set and evaluated their relative expression by Real Time PCR (qPCR).
This finding was confirmed by evaluating the expression levels of genes expressed during cardiac hypertrophy.
Cytotoxicity and transfection efficiency at different siRNA doses were monitored by evaluating REST expression.
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