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Such contradictory results could be influenced by differences in exposures, sampling, methodology, population genetics and/or environmental factors.
These gender differences in the prevalence of musculoskeletal complaints might be explained by differences in exposures to work-related physical and psychosocial risk factors [ 18].
Inconsistencies may be caused by differences in exposures to disinfection by-products and other water contaminants that can vary substantially by study population.
These changes in risks of migrating populations after living in a new environment suggest that the degree of difference by place and over time could be explained by differences in exposures and behaviours [ 3, 4].
The reasons for this disparity are not well understood, but some evidence suggests that responses to PM2.5 may be modified by differences in exposures, exposure measurement errors, composition of PM2.5, and underlying population susceptibility, including use of statin drugs which can offset inflammatory responses.
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This difference might be explained by differences in exposure for the different age groups (i.e., dissimilar based line of physiologic reserve among age group, different types of trauma and injury result in differential injury severity and risk of death).
Comparisons between studies are hampered by differences in exposure range, the pooling of different milk products as exposure, the method used for dietary assessment, the general dietary pattern, the prevalence of lactose intolerance, outcome assessment, and study size.
The incidence of hepatocellular carcinoma is 2- to 5-fold higher in men than in women, and it is uncertain if this difference is caused solely by the differing hormonal environments or is also influenced by differences in exposure to risk factors (2, 3).
This discrepancy may be explained partly by differences in exposure metrics.
Population-based half-life could be inflated by measurement error in population data, and its variance by differences in exposure.
The inconsistencies in associations of DDE with thyroid hormones cannot be explained by differences in exposure levels by study cycle.
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