Subsequently, we compared cell viability of drug-treated cells with those of vehicle-treated cells by using a standard cell viability assay.
Finally, exposure chamber dosimetry was assessed by measuring cell viability within a single chamber, and comparing cell inserts which were exposed to the same dilution of cigarette smoke.
These effects are also tested by human marrow stem cells (hMSCs) by comparing their cell attachment ability, cell viability and DNA transfection efficiency.
In the present study we investigated the difference in adherence of rFVIII to the cell membrane surface by comparing changes in cell viability and extent of phosphatidylersine (PS) exposure in apoptosis between rBHK-21host, rBHK-21Hsp70, and parental BHK-21 cells devoid of rFVIII expression (BHK-21native) during batch cell culture experiments.
The possible adverse effects of pressing on bacterial viability were investigated by comparing the viability of cells in tablets with those in formulated powders.
As the cell survival rates may be affected by drug treatments, we compared the cell viability of all protocols.
Efficacies of sensitization were determined by comparing the decrease in cell viability following combination treatments (i.e. drug in combination with TRAIL) to the reduction in cell viability as a consequence of the additive effect of single-agent treatments (drug alone + 10 ng/mL TRAIL alone).
Importantly, MDA231R280K acquired higher thermotolerance, indicated by higher cell viability, compared with MDA231 control cells.
In the absence of 5-FU, the knockdown of SphK1 in HCT116 cells by siRNA reduced cell viability by 35% compared with treatment with control siRNA, indicating that SphK1 normally maintains colon tumor cell survival.
Treatment of TSM1 cells and primary neurons with 400 µM MPP+ decreased by ∼60% and ∼80% the cell viability as compared to the control, respectively.
