The left angular gyrus was activated in another study comparing silent reading of high-frequency words with consonant strings (Joubert et al. 2004), but again, this activation did not survive a direct comparison between high- and low-frequency words.
These results suggest that Trp53 activation takes place in response to Ddb1 deletion but this activation does not account for the full spectrum of the phenotype.
Caspase 12 was triggered in ASFV-infected cells but this activation does not necessarily result in apoptosis, because the virus has the capacity to block ER stress at a later step.
This activation did not differ between the two groups when we examined regions of interest in the amygdala based on functional or structural criteria.
This activation does not occur in Whites processing White faces.
This activation does not require visual or auditory input.
This activation does not depend upon the mismatch repair complex and is cell cycle phase-dependent.
This activation does not depend on the presence of the ligands for Delta or Serrate.
This activation does not take place in mutants affecting cytoskeletal reorganisation such as Cdc42, Rac1 and Rac2 (Sampson and Williams, 2012b).
At an early postoperative stage, patients with left TLE without decline activated this posterior part of the hippocampal remnant more strongly (probably as a compensatory response) but this additional activation did not result in any further memory improvement.
