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The ethylene gas that some fruits emit inhibits bulb development.
Here we demonstrate that transcription factor AP-2ɛ is required for olfactory bulb development, specifically the establishment of appropriate neuronal lamination.
Alternatively, animals at the dolichocephalic end of the spectrum may have sufficient 'spare capacity' in the cranial vault to permit olfactory bulb development almost directly anterior to the frontal lobe.
This time course of bulb development is consistent with previous observations using γ-TIP as marker [ 10].
Studies have shown that KISS1R loss-of-function mutations contribute to nIHH by affecting GnRH secretion, but not olfactory bulb development or GnRH neuronal migration [ 15, 16].
In Lycoris sprengeri, DGE was performed to evaluate differential gene expression between bulbs and bulblets and determine the biological and molecular mechanisms underlying bulb development [ 5].
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In mammals, these proteins appear to regulate olfactory-bulb development and neuronal differentiation in the cortex [ 31, 32].
These findings therefore indicate that peripheral innervation contributes to bulb maturation late in development by promoting cell morphogenesis and differentiation.
Similar to other bulbs, the development of lily bulbs can be achieved by using photoassimilates for morphogenesis and the accumulation of reserve metabolites.
This argues against genetic interaction between Fgfr1 and Chd7 in olfactory bulb and hypothalamus development.
All these facts support the fact that carbohydrates are vital to bulb formation and development of bulbous ornamentals [ 8, 9].
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