Sentence examples for bronchial surface from inspiring English sources

Influenza virus antigen expression in lung sections was scored for bronchial surface epithelium, bronchial submucosal gland epithelium, bronchiolar epithelium, alveolar type I pneumocytes, and alveolar type II pneumocytes.

The fiber length is chosen to be compatible with an endoscopic use, so that the probe can be inserted in the working channel of a bronchoscope and its tip applied onto the bronchial surface of the patient mucosa.

Our data can provide better understanding about the association between polymorphism associated with β2-AR protein and the clinical markers of CF´s severity, and others studies about the large macromolecular complex are necessary to improve knowledge about the interaction of the proteins on the bronchial epithelial surface.

The eosinophils are selectively retained at bronchial microvascular surfaces by tumor necrosis factor alpha-induced and IL-4-induced upregulation of adhesive molecules.

Lf receptors are expressed on the apical surface of bronchial epithelial cells and this conception is utilized to achieve targeted drug delivery to the lungs (Elfinger et al. 2007).

Consistent with the histologic lung lesions, large amounts of SAα 2,3-gal were found on the surface of bronchial and bronchiolar epithelial cells of group NI puppies and were rarely found on tracheal epithelial cells.

AC inhalation increased surface SPH in bronchial epithelial cells of CF and CerS2-null mice (Fig 1C) with a concomitant reduction in ceramide levels (Fig 1D).

Expression of the DDR1 protein is located on the basolateral surface of human bronchial epithelium, where it interacts with type IV collagen with consequent activation of its tyrosine kinase activity.

Li and colleagues [ 106] have shown that DEP triggered Ca2+-influx through proteinase-activated receptor-2 (PAR-2) mediated activation of TRPV4 channels on the surface of human bronchial epithelial cells leading to increased expression of matrix metalloproteinase-1 (MMP-1).

We conclude that egression requires interaction of T-cell LFA-1 with either ICAM-1 or ICAM-2 on the basolateral surfaces of the bronchial epithelium.

Confocal microscopy showed that expression of ICAM-2 was found mainly on the apical and lateral surfaces on the bronchial epithelial cells (Fig. 2 D ′ H′), similar to that of ICAM-1.