Sentence examples for broader host response from inspiring English sources

An alternative approach would be to include cases that are identified based on clinical syndromes, such as severe sepsis, to identify genetic variation within proteins that are likely to play an important role in the broader host response to infection, regardless of the causative agent.

It is generally acknowledged that – apart from socioeconomic and demographic factors – a broader range of host response mechanisms, ranging from genetic factors, biological stress mechanisms and psychosocial processes, e.g. coping styles, social network of the patient, operate at the level of the individual, but have also an important influence at the population level [ 31, 33, 34, 36].

Additionally, functional differences in response to SIV or SHIV infection are also broad, and the host response to SIV influences the course of MTBC coinfection.

Post-transcriptional gene silencing is a part of a broad host defence response against nucleic acid invaders in most eukaryotic organisms, including filamentous fungi [ 34- 36].

This finding suggests that even susceptible genotypes have a systemic and broad host defense response mechanism that responds to X. fastidiosa infection, it does not prevent PD and must be augmented to achieve the resistance observed in 9621-67.

However, some of these agents will have broader effects on the host response.

Here I focus on novel autophagy bacteria interactions to illustrate these alternative outcomes, suggesting that autophagy should be viewed as having a much broader role in the host response to infection than only delivering bacteria to the lysosome.

This data set gives us a broad perspective for understanding host response and requires the appropriate bioinformatic analysis to interrogate key regulators of innate immunity.

A large number of genes involved in metabolism, tissue development, gene expression and the cell cycle were changed by MDV infection in all chicken lines which indicated broad similarities in the host response to MDV infection.

Grouping the genes identified in this study into broad functional categories, we identified several host response modifiers and host range genes that have sites under diversifying selection.

This aspect, in combination with its broad cell and tissue tropism, and limited viral host response has made it an attractive vector system for gene therapy.