Sentence examples for branching morphogenesis of the from inspiring English sources

Dean, C., Ito, M., Makarenkova, H. P., Faber, S. C. & Lang, R. A. Bmp7 regulates branching morphogenesis of the lacrimal gland by promoting mesenchymal proliferation and condensation.

A large surface area is created by extensive branching morphogenesis of the trophoblast-derived epithelium to create a villous network, called the labyrinth in rodents.

Thus, the activity of the stromally restricted homeodomain factor, Alx4, is required for normal branching morphogenesis of the ductal epithelia during pubescent mammary gland development.

In this review, we discuss the application of 3D culture models for studying branching morphogenesis of the mammary gland and the mammalian lung in the context of normal development and disease.

To date, only one study has addressed a role for microRNA in vascular development during branching morphogenesis of the lung, where miR-221 and miR-130a appear to have opposing effects on tube formation and migration of mouse foetal lung endothelial cells [25].

Previous studies revealed an inhibitory role for Bmp signaling on branching morphogenesis of the prostate.

At first sight these results on the mammary gland development of TGF-β1-null and TGF-β3-null mutant mice are difficult to reconcile with the experiments described above suggesting inhibitory roles for TGF-βs in the branching morphogenesis of this organ.

With FAK Src-family signalling identiFAK Src-familypotentially interesignallinghe fidentified screen, asd shown to beingquired for the branching morpotentiallyof tubules interestingnd round screen, there was suffinienthevidence to justifirstnal veroundation in intact tiscreen

In the developing rat lung, mesenchymal FGF7 expression is required for the induction of branching morphogenesis of distal lung endoderm and the maintenance of an alveolar epithelial cell phenotype [5].

Furthermore, because the global mechanisms controlling angiogenesis and the branching morphogenesis of various epithelial tissues appear to be highly conserved [ 1], our findings raise the exciting possibility that analogous mechanisms may also control SC identity in other systems.

In fact, a recent study attributed the ability of sVEGFR1 in rescuing the branching morphogenesis of developing vessels in VEGFR1−/− mutant embryonic cell cultures to sVEGFR1's role as a diffusible VEGF sink: that by diffusing away from the cell surface, it is able to alter the interstitial VEGF gradients as presented to endothelial tip cell filopodia of sprouting vessels [163].