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As (s_{mathrm {dynamic}}) decreases, the trajectory approaches the corresponding branch of fixed points, and KE cannot activate until this branch undergoes a saddle-node bifurcation (meeting the dashed fixed point branch in the figure) at the curve of jump up knees of the (V_{mathit {KE}}) nullcline (lower right cyan curve; also see Fig. 4).
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Near the tree branches in the figure, we also report 95% BCIs for the branch-specific relative rates r k.
We tested variable dN/dS ratios for PCSK9 among lineages, using the free-ratio model, which assumes a different dN/dS ratio for each branch in the phylogeny (Figure S1).
The major branches in this figure correspond to the well-known phylogroups.
However, in the interest of space we only show the major branches in this figure.
Lastly, the third branch, shown rightmost in the figure, starts from glucose and ends in CO2.
Interestingly, the three Cowden tumors, along with two supplementary tumors, lie on a separate branch, marked orange in the figure, characterized by over-expression of a group of about 84 genes (cluster e, Figure 1).
Unprotected wood communities were more heterogeneous in their composition as shown by a loose clustering and longer branches in the dendrogram (Figure 3).
Moreover, Q and T are the closest taurine branches in the tree (Figure 2) with a divergence time of about 40 50 ky (Table 3).
Regardless of the mechanistic explanation, one key result from our study was that molecular evolutionary changes were rather modest after 350 viral generations (sum total of 2750 generations, adding together the generations occurring on all of the branches in the tree; Figure 1); clones isolated at the tree tips sometimes contained only 4 substitutions separating them from the wildtype ancestor.
The four losses occur in the common ancestor of protosotomes (2; blue branch in Figure 5), in the lineage leading to C. elegans (1) and in the sea squirt (1).
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