Sentence examples for brainstem and the spinal from inspiring English sources

Fibres of the tract go to various regions of the brainstem and the spinal cord that organize movement.

The PAC1-Receptor mRNA are observed over wide area such as the olfactory bulb, the cingulate cortex, piriform cortex, the dentate gyrus of the hippocampus, the paraventricular nucleus, the ventromedial nucleus and the supraoptic nucleus of the hypothalamus, the cerebellum, the lateral paragigantocellular, the pontine nuclei and the vagal complex of the brainstem, and the spinal cord [38 41].

Disease progression was accompanied by the specific deposition of insoluble PS129-α-synuclein species, detected by biochemical and immunohistochemical methods throughout the brainstem and the spinal cord.

Disease onset in hA30Pα-syn mice was preceded by specific, progressive impairment of performance in two tasks —hindlimb extension reflex and rotarod tests— predictive of neuronal dysfunction in sensorimotor nuclei of the brainstem and the spinal cord.

The symptoms were accompanied by the deposition of PS129-α-synuclein but not PS87-α-synuclein in neuronal cell bodies and neuritic processes throughout the brainstem and the spinal cord; activation of caspase 9 was observed in 5% of the PS129-α-synuclein-positive neurons.

In contrast, there was extensive PS129-α-synuclein immunoreactivity in various regions of the brainstem and the spinal cord in end-stage symptomatic hA30Pα-syn mice with or without Parkin, but not in their non symptomatic littermates (Figure 4A, B and Figure 5A, B).

Our results suggest that the development of tolerance in the production of antinociception in mice forced to the repeated CWSS may be, at least, due to the reduction of DOR number in the brainstem, midbrain and the spinal cord regions.

In the opioid receptor binding study, Bmax of delta opioid receptor (DOR) was decreased in the brainstem, midbrain and the spinal cord areas by both a single and repeated CWSS, without altering Kd value.

They arise most frequently in the cerebellum, followed by the optic pathways, where they occur as sporadic tumors or in the context of neurofibromatosis type I, followed by the brainstem, thalamus and the spinal cord.

Finally, the neuropathic pain alters function and activity in the descending controls, which relay information from higher brain centers via the midbrain and brainstem to the spinal cord.

PA can arise anywhere in the CNS, although it most frequently occurs in the cerebellum (42 %), followed by the supratentorial compartment (36 %), the optic pathway and hypothalamus (9 %), brainstem (9 %), and the spinal cord (2 %) [ 9].