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Brains were evaluated by histology with hematoxylin and eosin and immunohistochemical reaction for glial fibrillary acidic protein (GFAP).
The brains were evaluated for histological evidence of damage from CO.
Treatment continued for two weeks at which time brains were evaluated for cell survival and the abundance of graft-derived neurons.
In addition, mice brains were evaluated for microglial activation and BBB dysfunction.
bBlue Jay brains were evaluated for viral load in both cerebellum and cortex.
To assess the effect of bacterial meningitis on brain damage, the brains were evaluated histomorphologically.
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Mean amyloid beta 42 present in mouse brains was evaluated by mouse A β42 ELISA kit.
The histological demonstration of the fibre tracts was also correlated with high-resolution post-mortem MRI and the variability of their trajectory and position in different brains was evaluated.
The incidence of seizures, mortality, neuron counts, neuropathology and apoptosis in specific regions of the brain were evaluated.
The effect of treatment on demyelination and lymphocyte recruitment to the brain were evaluated.
Patients with subsequent disease progression in the brain were evaluated for and treated with SRS when appropriate.
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