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In addition, [14C]phenylacetate uptake in the rat brain was compared by autoradiography with that of [14C]acetate following the injection of fluorocitrate, a selective glial toxin, into the brain.
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Then, the pharmacokinetics of these candidate compounds in the rat brain were compared by ex vivo LC-MS/MS assays.
The effects of different fractionation schemes on the normal brain were compared by calculating the biological-equivalent dose (BED) (Fowler, 1989) using a normal tissue alpha/beta ratio of 3 (Lee et al, 1998).
The fluorescent concentrations in mouse brains were compared by one-way ANOVA followed by Newman-Keuls post-hoc test.
The spatial and temporal patterns of Nav1.1 expression in Scn1afl/fl and Scn1afl/fl, Emx1-Cre brains were compared by immunohistochemistry.
The severity of HSP27 immunoreactivity in neurons and glial cells in the different brain regions in all four diagnostic groups was compared by Kruskal-Wallis test, with post hoc Mann-Whitney testing where significant.
To determine if CNS-resident cells contribute to TIMP-1 mediated control of MMPs during Toxoplasma infection microglial populations isolated from naive and infected whole brain tissue were compared by flow cytometry for TIMP-1 production.
To further test the tumor specificity of scL nanocomplex, Cy5-ODN uptake in various types of normal brain cells (i.e., neurons, astrocytes and microglia) and brain tumor cells were compared by flow cytometry in mice bearing intracranial T98G GBM xenografts.
Kaplan-Meier survival curves for the first episode of LRTI were plotted according to the severity of brain atrophy, and data were compared by the log-rank test.
Groups of patients were compared by calculating standardized incidence ratios (SIRs) for brain tumours in offspring.
Two groups were compared by t-test.
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