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Another region of the brain that may mediate the effects of BDNF signaling on food intake is the brainstem.
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A study of 18 abstinent alcoholics and 17 healthy control subjects acquired fMRI data during an attentional Stroop task (Schulte et al. 2012) and revealed abnormal synchrony in networks in the brains of abstinent subjects that may mediate between the top-down executive control and bottom-up appetitive drive networks.
It is clear that early detection of AD will be facilitated through continued investigation of the relationship among the behavioral manifestations of geriatric depression, the brain changes associated with the AD pathological process, and factors that may mediate this association.
The correlation of these volumetric reductions with poor neurocognition indicates that these brain regions may mediate higher neurocognitive functions implicated in schizophrenia.
A new study in rats suggests that hormone levels in the brain may mediate alcohol's potency, giving females a leg up at certain times during their hormonal cycle.
We conclude that acj6 neurons in the central brain may mediate a general escape response circuit, which is triggered by a variety of alarm signals.
One molecule that may help mediate the relationship between stress and alcohol consumption is brain-derived neurotrophic factor (BDNF).
New evidence does suggest that T2 may mediate the effects of antidepressant therapy in the brain.
The idea that neurotrophins may mediate the neuroprotective effects of exercise in the brain comes from several studies where increased physical activity elicited increased expression of BDNF [ 14, 15].
We reasoned that oxidative‐stress may mediate C/EBP‐ δ upregulation, thereby stimulating serpina1 production in the brain of Cln1 −/− mice.
Some evidence from animal studies suggests that testosterone may mediate cognitive differences between the sexes via organisational effects on the brain.
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