Sentence examples for brain parenchyma with a from inspiring English sources

PARVB, LIMS1, PRCP, PTRF and FSTL1 were found in capillaries in brain parenchyma, with a similar pattern as CDH5.

Meningiomas usually appear hyperechoic, compared to normal brain parenchyma, with a homogeneous pattern and a granular aspect probably due to psammomatous bodies and fine trabeculature (WHO I-II) or with numerous hypoechoic areas of necrotic degeneration (WHO III) [ 39, 40].

On standard US B-mode imaging glioblastoma (n − 47) appeared all hyperechoic compared to brain parenchyma, with a heterogeneous appearance composed of multiple well-defined nodular areas and others with diffuse margins.

Then 5 × 10 MSCs in 6 μL of PBS were injected carefully into brain parenchyma with a Hamilton syringe within 10 min. The luciferase activity of MSC-Fluc was visualized and quantified by a Xenogen IVIS-100 system (Caliper Life Sciences, Hopkinton, MA, USA).

A cerebral infarct was defined as a defect of the brain parenchyma with a signal intensity that is isointense to that of cerebrospinal fluid on all pulse sequences (i.e., FLAIR and proton density/T2-weighted) and that has a minimal diameter of 4 mm, with the exception of infarcts in the cerebellum, which had no size criteria.

WMLs were defined as areas of brain parenchyma with an increased signal on the T2-weighted scans but without significant volume loss.

Animal models can also be used to track CD163 immunoreactive peripheral macrophages and their probable entry into the brain parenchyma with or without a breach of the BBB.

Second, high levels of soluble Aβ40 in brain parenchyma, with up to a threefold increase, distinguish demented patients with AD from non-demented patients, even though the non-demented patients may have high numbers of insoluble Aβ plaques in their brains [ 62].

When bacterial (and some fungal) microorganisms cause a leptomeningitis, a clinical imperative is to prevent this meningitis from evolving into a meningo-encephalitis or cerebritis that leads to significant injury, necrosis, and inflammation of the brain parenchyma with far more serious neurologic morbidity than an infection isolated to the subarachnoid space.

Given that neuronal ER calcium can flow freely [40], [41], the partitioning caused by fission may serve to limit the release of calcium from the ER lumen under conditions when e.g. part of a neuron's dendritic arbor lies within a region of the brain parenchyma with excess levels of extracellular glutamate.

There was a reversal of the Aβ40/Aβ42 ratio in brain parenchyma with advancing age. Figure 4 shows the accumulation of Aβ40 and Aβ42 plotted with the changes in CSF turnover with age.