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How does the BTT address the apparent problem of "brain interventions," such as LSD, microelectrodes, or brain tumors?
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Our finding of relationships between impairment and FCR or FA asymmetry raises the possibility that such measures could be used to tailor individual therapeutic interventions, such as brain stimulation approaches (Hummel et al., 2005; Mansur et al., 2005).
Invasive neurotechnology interventions such as deep-brain stimulation (DBS) involve the alteration of the patient's neural processing by electrode-delivered electrical impulses.
In doubtful cases, always correlating with the clinical signs of brain death, modifications of the waveforms with interventions, such as osmotic therapy, may allow practitioners to discard the CCA diagnosis given the reversibility of the case on follow-up examinations.
First, we recognize that the EEG signals, recorded during experimental interventions (such as global ischemia brain injury reported here), are inevitably nonstationary.
The next goal is functional genetic neuroimaging – studying changes in gene expression in the brain across time, for example, during development or following interventions such as drugs or cognitive tasks.
This is important when defining optimal blood and brain glucose levels with the aim of avoiding deleterious consequences of routine therapeutic interventions such as insulin administration.
EEG could reveal different properties of these brain networks, such as timing of event processing, and may be more amenable than fMRI to active interventions such as neurofeedback.
Among secondary injuries, cerebral edema is of special significance, as it can greatly aggravate brain damage and is the main condition related to increased ICP, excluding conditions potentially leading to surgical interventions, such as hematoma and contusion.
But, it is plausible that some interventions, such as a dose of hallucinogen or maybe some carefully placed microelectrodes, could trigger such brain events, quite apart from the connection of those brain events to other events in the external world.
We've come to view ADHD more as a chemical problem in the brain that should be treated using stimulants, he explained, rather than considering potential psychological causes and behavioral treatment interventions, such as psychotherapy.
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