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The effects of PCBs/dioxins on the brain have been interpreted in several ways.
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In MS, acetate accumulation in brain has been interpreted in terms of reduced acetate incorporation into lipids, and the latter was suggested to play a role in altered myelin stability seen in MS [27].
The observation of increased density of cortical minicolumns in autistic brains has been interpreted as suggestive of a skew in the ratio of short U fibers to long association fibers (Casanova et al. 2002).
The different effects of practice on brain function have been interpreted in terms of reorganization vs. redistribution of activity [40], changes in skill or strategy underlying task performance [41], item-specific or task-skill effects [42], or improved task proficiency [22], [43], [44].
These results have been interpreted as an increase in P-gp function in the brain of schizophrenic patients.
Such differences have been interpreted by hypothesizing that real and pantomimed actions are governed by different visual brain pathways [4].
"Chairman Steele regrets the way his comments have been interpreted.
Furthermore APP-cleaving secretases and Aβ have been localized to autophagosomes and the accumulation of autophagosomes in AD brains and APP/PS1 mice has been interpreted as evidence that autophagy could promote AD pathology [10].
This has been interpreted as an indicator of brain shrinkage after the onset of the brain process associated with schizophrenia since the skull in an adult or adolescent is unlikely to contract.
The N400 has been interpreted as a default brain response indexing the retrieval of semantic information for a stimulus given preceding context (Brouwer & Hoeks, 2013; Kutas & Federmeier, 2011).
This has been interpreted in various ways.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com