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In the last two decades, there has been a considerable amount of fMRI studies for adult BD pointing to a relevant role of brain functional abnormalities in the pathophysiology of the disease.
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We aimed to clarify brain functional abnormalities related to motor response inhibition in schizophrenia by using event-related fMRI in combination with a Go NoGo task designed to control for non-inhibitory cognitive processes involved in task performance.
The present study aimed to identify brain functional abnormalities specifically related to motor response inhibition in mania by using event-related fMRI in combination with a Go/NoGo task designed to control for extraneous cognitive processes involved in task performance.
Thirdly, these results should be tested in a study which compares brain functional abnormalities of a same person with different mood states.
Indeed, structural and functional abnormalities in brain areas involved in object representation have been reported in schizophrenia [ 2, 13- 15].
Whatever the etiological factors, there exists abnormality in perfusion and functional abnormalities in brain of the children with craniosynostosis [ 10, 15].
Among these, some of the most important techniques include anatomical magnetic resonance imaging (MRI) and functional MRI (fMRI), which can reveal anatomical and functional abnormalities in brain development.
On a more fundamental level, studies on neurobiological effects of CAN point to structural and functional abnormalities in brain development [ 59, 85, 86].
Gaining a better understanding of the nature of the alterations in brain functions that occur in ASD could be of vital importance to try to identify the link between anatomical and functional abnormalities in the brain of persons with ASDs and their behavioral phenotypes.
Recent MRI studies have shown structural and functional abnormalities in the brain of cluster headache (CH) and migraine (M) patients.
Previous studies have suggested that structural and functional abnormalities in the brain network may contribute to resistance to antidepressant treatment in depressed older patients without dementia [26, 27].
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com