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To maintain adequate brain function and neuronal integrity, cerebral blood flow (CBF) under physiologic circumstances is autoregulated within a wide range of cerebral perfusion pressure.
Poor nutrition leading to dietary deficiencies of zinc and/or other essential dietary nutrients such as selenium, amino acid methionine, and essential fatty acids can disrupt metabolic processes and impair brain function and neuronal plasticity by exacerbating heavy metal neurotoxicity [ 1, 16- 18].
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To examine our hypothesis that MDA is one of the major substances in the brain leading to fatigue, the influences of MDA on brain functions and neuronal encodings in red-eared turtle (Trachemys scripta) were studied.
Neuronal connectivity and neuroplasticity have an impact on brain functions and neuronal organization throughout adulthood and environmental factors will influence these processes.
The central hypothesis examined in this issue is that insulin resistance promotes maladaptive brain function and contributes to reduced neuronal plasticity, potentially accelerating brain aging.
The cerebrospinal fluid, its composition, circulation and absorption, has multiple roles in both normal and abnormal brain function and is closely associated with the neuronal interstitial fluid.
In this review, we describe the important potential of DHA to preserve neuronal and brain functions and classified its numerous molecular and cellular effects from impact on membrane lipid content and organisation to activation of signalling pathways sustaining synaptic function and neuronal survival.
Most approaches to brain function consider neuronal firings and synaptic transmissions as fundamental information states (e.g. 'bits') in computational networks of neurons.
It has been utilised to study brain function following neuronal activation, as well as for the detection of changes of perfusion occurring during brain pathology, maturation and aging [27], [28].
DHA is important for the structure of the neuronal membranes, while EPA plays a role in brain function by regulating neuronal signaling, neurotransmitter uptake, and the activity of phospholipase enzymes.
DM areas are independent of task active areas, and suspension of baseline DM brain function allows reallocating neuronal resources to task-relevant brain areas.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com