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Brain edema could be due to both vasogenic and cytotoxic changes.
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Diffuse subarachnoid hemorrhage and edema could be seen within the brain parenchyma, which showed a loss of integrity.
Diabetic macular edema could be managed with ranibizumab in either group.
This edema could be the cause of the herniation.
Controlling brain edema might be the most important therapy to help HSES patients survive.
Controlling brain edema might be the optimal therapy to help HSES patients survive.
Glioma-related edema (GRE), especially peritumoral brain edema (PTBE), is seen commonly in the clinic.
Brain edema formation, is one of the most dangerous consequences of ischemic brain injury [ 38].
The peritumoral brain edema index was defined as volume of tumor + volume of edema/volume of tumor.
Alterations of BBB permeability with subsequent brain edema formation are common features of septic encephalopathy.
Peritumoral brain edema has been observed in 40 to 60% of all meningiomas [ 22].
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