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Sepsis-associated encephalopathy (SAE) is a state of acute brain dysfunction in response to a systemic infection.
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In addition, anti-inflammatory response is of pivotal importance in the evolution of sepsis [ 16], and can contribute to brain dysfunction in animal models of sepsis [ 17].
Traumatic brain injury (TBI) causes brain dysfunction in many patients.
Introduction: Delirium is a common manifestation of acute brain dysfunction in critically ill patients.
The patient developed bradypragia and higher brain dysfunction in mid-October mid-October 2011
Detection of acute brain dysfunction in ICU is based on repeated daily neurological examination.
However, phenotypic analysis is not sufficient, particularly for brain dysfunction in neurogenetic diseases.
Diagnosing brain dysfunction in a patient with sepsis implies a systematic diagnostic approach of all potential factors, in addition to sepsis, that can contribute to aggravate or prolong brain dysfunction.
The diagnosis of brain dysfunction in sepsis relies essentially on neurological examination and neurological tests, such as EEG and neuroimaging.
To investigate cortisol levels in brain dysfunction in patients with severe sepsis and septic shock.
The findings suggest more pronounced organic brain dysfunction in AUDS- than in MDE patients.
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