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We hypothesized that diabetes may cause 'silent' changes affecting astrocytic communication and metabolite trafficking via gap junctions may alter interactions among astrocytes, neurons and endothelial cells (i.e. the neurovascular unit), thereby contributing to the slow, progressive brain dysfunction in diabetes.
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Traumatic brain injury (TBI) causes brain dysfunction in many patients.
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Detection of acute brain dysfunction in ICU is based on repeated daily neurological examination.
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To investigate cortisol levels in brain dysfunction in patients with severe sepsis and septic shock.
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