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Local brain cooling is a way to treat traumatic brain injury and prevent secondary brain injury.
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In intubated stroke patients, brain cooling is faster during CI than during NPC.
First study showing the effect of CI on brain temperature Brain cooling is faster during CI (4°C, 2 L at 4 L/hour) than during NPC.
In intubated stroke patients brain cooling is faster during active treatment with CI (4°C, 2 L at 4 L/hour) than during NPC.
After the stabilization period, intranasal brain cooling was initiated by blowing cold air (−3 ± 2 °C) at a flow rate of 40 50 L/min into both nostrils for 50 60 min.
in the first experiment, nasopharyngeal brain cooling was initiated by blowing room temperature air from the hospital medical air outlet, at a flow rate of 14-15 L/mintonto both nostrils for 60 min. the brain was then allowed to gradually rewarm to the baseline temperature.
The normal physiological mechanisms of brain cooling are heat loss from the upper airways and through the skull, and these can produce selective brain cooling.
Intranasal brain cooling was achieved by connecting two nasal catheters (made from polyvinyl chloride, PVC) to the tube from the cold air outlet of a vortex tube.
Following rewarming, the second series of measurements and brain cooling was performed in the same manner as the first one but blowing cold air (-7°C) at the same flow rate.
Four babies (three by day and one at night) were treated for hypoxic ischaemic encephalopathy and in all cases the cerebral function analysis monitor was normal and brain cooling was not required.
Cool is a ghetto.
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