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The relation between trial-by-trial differences in RT and brain activation was modeled in five different fMRI datasets spanning a range of experimental tasks and stimulus modalities.
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Whole-brain fMRI activation was modeled as the contrast of temperature stimuli (those temperatures perceived as producing warmth versus mild pain versus moderate pain against a fixed baseline).
At the first level, brain activation was estimated for each fMRI task at each voxel under the general linear model framework.
Brain activation was measured using a block design paradigm.
Neuromagnetic brain activation was elicited by a finger-tapping motor task.
The fMRI results revealed that their brain activation was at a level never documented before.
20 21 Ipsilateral brain activation was also observed.
Brain activation was compared within patients under either drug condition.
The calibration of the hemodynamic model that describes changes in blood flow and blood oxygenation during brain activation is a crucial step for successfully monitoring and possibly predicting brain activity.
Group differences in brain activation were identified using analysis of covariance.
It was observed that Abeta was effectively cleared from the brain parenchyma and brain microglial activation was reduced in long-term therapeutic immunization of an AD mouse model bearing the DRB1∗1501 allele [ 45].
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