Hydroethanol extracts of B. africana were reported to decrease bovine phospholipid damage (IC50 = 78 μg/ml) [ 7] and although no IC50 was obtained in the present study, a 43% inhibition of lipid peroxidation was seen for the polyphenolic-rich fraction at 20 μg/ml.
In addition, Singhal and coauthors described that the residual elastic strains in the hydroxyapatite phase decrease markedly with increased X-ray irradiation of cortical bovine bone, indicating damage at the hydroxyapatite-collagen interface [ 42].
Tew et al. [9] also found in bovine cartilage explants damaged by cutting that apoptotic cells detected by TUNEL staining remained in the tissue for 20 days following injury.
Frank et al artificially damaged bovine articular cartilage by trypsin digestion and applied impedance analysis to the treatment site.
A solution of poly methyl methacrylate- co-methacrylic acid- co-ethylene glycol dimethacrylate) microgels functionalized with glycidyl methacrylate was injected into the tissue free space of damaged bovine IVDs.
In this work, a novel, sensitive and inexpensive method for direct electrochemical detection of bovine serum albumin (BSA) damage in aqueous solutions is described.
It was concluded that continuous loading for >40 min causes creep damage in bovine TMJ disc, and that prolonged sustained tension affects the recovery of joint homeostasis.
One motorist collided with a bovine and sustained significant damage to his vehicle.
Although NAD(P H protects bovine catalase against oxidative damage by its peroxide substrate, the catalytic role of the nicotinamide cofactor in the function of this enzyme has remained a biochemical mystery to date.
Oxidative stress by ROS must be one of the causes which may induce lipid peroxidation and/or organelle damage in bovine oocytes [ 112].
