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The intracellular distribution and location of BSA NPs in both types of tumor cells were observed by confocal laser scanning microscopy, as shown in Fig. 2.
Both types of tumor exhibit sigmoid curves which differ in a very characteristic way in profile.
In contrast, synthesis of the CS chains suggests changes in epimerization and sulfation of the C4 and C2 in both types of tumor.
IFN- β and Noxa were significantly induced by reovirus via the IFN- β promoter stimulator-1 (IPS-1) signaling in both types of tumor cells.
For the dose of 36 C/cm3 there were no significant differences between both types of tumor (P > 0.05); however, for the other doses there were significant differences (P < 0.05).
Some proteins were found in both types of tumor; whereas some proteins were only found in the tumors with low metastatic potential and some were only found in the highly metastatic tumors.
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Finally, a control group without treatment served to monitor the normal in vivo growth and radiotracer uptake of both types of tumors.
In both types of tumors, the intra-nuclear architecture of chromatin was considered to affect the susceptibility of genomic segments of the p16 locus to DSBs.
In order to assess the therapeutic effect of the different medications tested on both types of tumors, the uptake of each treated group was compared to the values from the untreated group obtained with the same tracer.
Broken DNA ends were joined by non-homologous end joining (NHEJ) repair in both types of tumors, however, microhomologies of DNA ends were preferentially utilized in the joining in solid tumors but not in lymphoid leukemia.
Both types of tumors commonly occur concurrently.
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