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In humans, by contrast, both ciliated and goblet cells can be labeled with MAA/MAL-I and SNA [19], [20], indicating that these two epithelial cells have both types of influenza viral receptors.
Indeed, recent results indicate that this approach has the potential to identify PPI inhibitors that prevent viral polymerase activity and replication of both types of influenza viruses (Kessler et al., unpublished results).
Only a few risk factors were associated, significantly or at least in a trend, with all outcomes of interest for both types of influenza (table 5).
Some African countries, including Kenya, Mauritius, Senegal, South Africa, Tunisia and Uganda have also reported co-circulation of both types of influenza virus during 2008 [ 3, 4].
Using RT-PCR as the gold standard technique, the results showed that 6 (7.5%) samples were positive for influenza viruses and 1(b)), among which only one sample (1.25%) was positive for both types of influenza viruses) as a coinfection.
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Both types of licensed influenza vaccines rely on fertilized chicken eggs as substrates for production [4].
Both the M and NS genes are less differentiable between these 2 types of influenza A viruses.
Therefore, both proteins are considered key targets for vaccination and are included in all types of influenza vaccines (although antibodies directed against NA are not considered neutralization antibodies).
There are three types of influenza virus, namely, A, B, and C. Influenza A (both H1N1 and H3N2 subtypes) and influenza B viruses circulate among the human population each year and are the causative agents of seasonal flu.
Type A is one of the three major types of influenza viruses (the other two being types B and C).
But the insurance mechanism is limited to certain classes of infectious diseases most likely to cause major outbreaks, including several types of influenza, respiratory diseases such as SARS and MERS, and other deadly viruses including Ebola and Marburg.
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