Building materials emit a myriad of reactive constituents and secondary products (derived from initial constituents).
Postmortem examination showed characteristic necrotic lesions with marked central lysis of tissue, reactive gliosis, and secondary demyelination in the underlying white matter.
Care was symptom-focused, episodic and reactive, in primary and secondary care settings, and was consequently poorly aligned with the long-term, multidimensional needs of this patient group.
The 1999 ASPEN model incorporated meteorologic data, emissions data, and determinants of ambient air pollutant concentrations such as: rate, location and height of release, reactive decay, deposition, and secondary formation [ 24].
Some areas showed confluence of many recent and old necrotic lesions, leading to an extensive destruction in all cortical laminae, reactive fibrillary gliosis, and secondary demyelination in the underlying white matter.
This suggests that h+ and O2 − are the dominant and secondary reactive species causing the dye degradation, respectively.
The active species trapping experiments implicate OH and O2− radicals as the respective primary and secondary reactive species responsible for oxidative photodegradation of organic pollutants.
The acquired forms may be primary (immunologic) and secondary (reactive) [ 2].
This reduces the paracrine signaling cascade mediated by Wnt4, inducing TCF3-dependent repression [ 52] and/or primary downregulation [ 47] of canonical Wnt signaling and secondary (reactive) upregulation of Notch signaling in basal stem/progenitor cells.
Oxidative stability of flaxseed oil was also examined by measuring peroxide values and thiobarbituric reactive substances to evaluate primary and secondary oxidation, respectively, at different processing steps.
