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Firing rate patterns and statistics of both populations were analyzed over a range of the inter-population connectivity values.
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Health care resource utilization for both populations was analyzed separately in public and private hospitals.
All X-chromosome and mtDNA markers showed a significant correlation (p < 0.05) with Y-chromosome SNPs both when 9 or 11 populations were analyzed.
For both OX4319L-Pxy and OX3097D-Bol, three replicate populations were analyzed with initial transgene frequencies of 0.25, established by crossing transgene-heterozygous males with wild-type females; this represents the maximum starting allele frequency for a female-lethal transgene in the absence of artificial releases of homozygotes.
After 2 weeks, CD45.1+ populations were analyzed by flow cytometry as described above.
Cell culture populations were analyzed by flow cytometry using a BD Accuri C6 (BD Biosciences, San Jose, CA).
Myeloid cell populations were analyzed in LysMCre/+ Ifngr1fl/fl and control mice.
Myeloid and dendritic cell populations were analyzed by flow cytometry 4 days after PBS, r3LCMV or WT LCMV infection.
Treg cell populations were analyzed by flow cytometry in NPs and control tissues from GC-treated CRS patients and controls.
(b-e) Myeloid cell populations were analyzed by flow cytometry in LysM-GFP mice infected with r3LCMV or PBS, or in C57BL/6 (B6) mice as a control.
(d,e) Myeloid cell populations were analyzed by flow cytometry at 4 and 6 days after PBS, r3LCMV or WT LCMV infection.
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