With the increase in salt concentration, PEPC was significantly up-regulated (F3,40 = 153.8, P< 0.0001) (Fig. 10A), while RUBPC was significantly decreased (F3,40 = 334.6, P< 0.0001) (Fig. 10B) in both types of plants compared with plants grown without salt.
The net photosynthesis rate was measured only for the second experiment with cv Bonus, and it was higher in both single and mixed inoculated plants compared with the control in the two water regimes (Fig. 2a, b).
At this stage, AtHB12 transcripts were 4-fold higher, both in AT7 and athb7 plants compared with WT plants.
The NP exposure was associated with increased bacterial growth, both in local and distal leaves of infected plants, compared with the control.
The stronger activation of both PR1a and PBZ1 in mutant-inoculated plants compared with wild-type controls suggested that ΔpdeH mutant can still elicit plant defenses, which may be involved in attenuation of the virulence of the ΔpdeH mutant.
However, the only available biological replicate at D28 (n = 1) suggested a lower amount of soluble proteins at D28 in LN plants compared with HN for both genotypes.
An increased level of antioxidative enzymes involved in H2O2 detoxification, such as superoxide dismutase, catalase, ascorbate peroxidase, and peroxidase was observed in both leaf and root tissues of Pb treated plants, compared with the control.
The effect of the B allele on time to bolting was also demonstrated by the earlier (∼3 days; P < 0.01) bolting of BB plants compared with Bb plants in both the 950619 and 960701 populations.
Both species of bacteria were more abundant in inoculated plants compared with the mock treatment, and there was an increase in bacterial numbers seven days after inoculation.
As a result, both root and leaf hydraulic conductivities were significantly reduced in PsPIP2 1-silenced PsPIP2 1-silencedith control plants.
