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In fact, endosomes and MVBs can be connected to the trans-Golgi secretory pathway [3], thus both pathways could be involved in vesicular polysaccharide export in fungi.
In our study, the sequences associated with both pathways could be identified (Table 4).
This suggests that both pathways could be altered in sima loss-of-function strains.
Both pathways could be operationalized at the same time and in response to known genetic and environmental triggers.
As such, effects on both pathways could be acting on chemokine induction in direct and indirect ways.
It is conceivable that unemployment may result in the onset of problem drinking (Claussen, 1999) or vice versa, (Kriegbaum et al., 2010) although both pathways could be operating simultaneously.
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Both of these pathways could be involved in the regulation of p27 in human cancer favoring cell proliferation and tumor progression.
The GSK3β-dependent TNFα resistance and NF-κB activation [8] in adherent leukemic cells suggest that both extrinsic and intrinsic apoptotic pathways could be regulated by α5β1 integrin engagement.
Therefore, both FoxO3 and GSK-3β pathways could be regulated during PDGF-AA- and bpV pic -induced hSKP senescence alleviation.
These discoveries provide additional evidence that common pathways could be utilized both in the acquisition of pluripotency and in tumorigenesis.
Thus, a therapy targeting the PDGF-B signaling pathway could be effective for intestinal-type gastric carcinoma, whereas targeting VEGF-A or both VEGF-A and PDGF-B signaling pathways could be effective for diffuse-type gastric carcinomas.
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