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However, the presence of FTEn luteal samples in both of these cluster groups indicates that BRCA mutation status does not affect the expression of PR-A-responsive genes.
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Both of these clusters likely belong to a single operon, considering the very short intergenic regions and the overlap of coding sequences in the organisms.
If yes, the merging begins and both of these clusters first turn their states into Merging, the cluster with more sensor nodes will be the initiative one and its cluster head will record the information of the new merged cluster.
Both of these clusters contain genes conferring specialized or accessory functions beyond those conferred by the core degradation pathways.
In most tumours there was a coordinated decrease in both of these clusters, but we observed that in some tumours these facets of phenotype change were uncoupled.
Both of these clusters comprise CAPRIN1 and MOV10, while they do not share FUS, FXR1, TAF15 and C22ORF28 (see Table 2).
When taking into consideration two large PFGE-E clusters, it is clearly demonstrated that strains from both of these clusters were almost regularly distributed among the MP-E types as well as differentiated as singletons.
The Aux/IAA transcription factor family was enriched in both of these clusters, and the auxin-associated functional group was enriched in K6 (Fisher's test, p-value ≤ 0.05) (Additional file 5).
Most of them were grouped in Clusters A and C. The expression levels of the genes in both of these clusters reached a minimal expression level at 24 hpi followed by either a minor (Cluster A) or major (Cluster C) recovery by 72 hpi (Table 1).
The reads mapped to both strands of these clusters, but with strand-specific differences in their downstream nucleotide composition (Additional file 1 Figure S2).
In contrast, clusters of starvation-induced genes detected by Pyxis in the Radonjic et al. dataset rarely overlap clusters of genes repressed by starvation in the Gasch et al. dataset – just 21 genes are found in both sets of these clusters.
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