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The regulation of protein trafficking is known to be involved in both normal function and dysfunction of the nervous system.
Because this is likely to affect other cell types such as stromal, immune, or inflammatory cells, it confounds our understanding of the role of the EP receptors in keratinocytes, in terms of both normal function and NMSC development.
It is also noteworthy that astrocytes can couple to neighboring astrocytes through gap junctions (Fig. 2a) formed by connexins, and that the gap junctional coupling of astrocytes into multicellular networks may play a role in both normal function and CNS disorders [ 162, 212].
Studies in knockout mice models further indicate that while the TRα1 isoform is not an absolute requirement for normal auditory development, there is a synergism between the α and β thyroid receptor subtypes for both normal function and the anatomical development of structures within the cochlea.
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Mitochondrial ADP, ATP carrier can be implicated to both normal functions and cell death [ 24].
Middle-ear anatomy is integrally linked to both its normal function and its response to disease processes.
By continuing to characterize the mechanisms underlying both the normal function and pathological degradation of oral and maxillofacial structures, improved tissue-engineering constructs can be designed with the unique local environment of this region in mind.
Independently from a role in pathogenesis, a comparison between full-length ataxin-3 and isolated Josephin can help us to understand the relative influence of different and distal regions of the protein both on its normal function and on disease pathogenesis.
For example, in the adult mouse brain, Dnmt1 and Dnmt3a are both required for normal function and neural plasticity.
The present results also suggest that phasic activity changes in the EPN through the cortico-striato-EPN direct pathway are fundamental to both normal functions of the BG and the pathophysiology of movement disorders.
Glucose is an especially important substrate for the heart [ 17], and influences both normal cardiac function and the response of the heart to ischemia that promotes glucose uptake and decreases the utilization of free fatty acids by the human heart [ 18].
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