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It has been suggested that post-translational modifications of one or both molecules may be required for complex formation[5].
A 6-days treatment with either TA or hTA in the 0 to 20 µM range of concentration did not prevent PrPSc accumulation in chronically-infected MovS6 cells as shown by Western blot analysis (Figure 2, panel a and Figure S2, panel b) suggesting that both molecules may be inactive against these mammalian prions in the tested range of concentration.
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Combinatorial treatment modulating both apoptotic and costimulatory molecules may be more effective for reinforcing host immunity and eradicating tumors.
Contraceptives releasing both antimicrobial and contraceptive molecules may be one avenue towards a contraceptive microbicide that potentially could also improve compliance and product adherence (www.ipm-microbicides.org).org
Some edge case compounds with significant amounts of both dissolved and undissolved molecules may be described differently by different solubility tables.
For both regards, investigations of the interaction between these molecules may be of interest and contribute to elucidate biological processes.
The overexpression of TLR7 MyD88-dependent signaling molecules may be a common pathogenic mechanism for both AOSD and SLE.
Therefore, small molecules may be an alternative.
The molecules may be natural; the processes for making them are normally not.
In addition, levels of specific immune system signaling molecules may be altered.
The monomer molecules may be all alike, or they may represent two, three, or more different compounds.
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