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The two main findings are as follows: In both models, we find that migration has a direct positive impact on the ability of a single mutant cell to invade a pre-existing colony.

In both models, we find extensive epithelial hyperplasia, especially in the distal bronchioles and adjacent alveoli, which progresses to carcinoma in about half the mice over a period of weeks.

Also in both models, we find a significant impact of idiosyncratic effects on the market outcomes.

In both models, we find a significant impact of idiosyncratic effects on the market outcomes.

In both models, we find that migration has a direct positive impact on the ability of a single mutant cell to invade a pre-existing colony.

Similar(55)

In both models, we found significantly increased CO4-MMP levels in rats with fibrotic livers compared with controls.

In both models, we found that human NSCs survive, migrate, and differentiate within the host hippocampus leading to significant improvements in cognitive function.

In both models, we found a relevant role of hERG1 channels, which could be traced back to a hERG1-dependent control of angiogenesis.

Using the same experimental protocol for both models, we found that the adenine model provokes massive phosphaturia and a more severe form of bone disease, although we observed no significant differences between the two models in terms of VC.

Further we noticed that in both estimated models, we find a significant impact of idiosyncratic effects on the market outcomes.

Using linear regression models, we find both, decreasing Euclidean distances to the nearest green space and increasing spatial coverages of UGS significantly contributing to SRH.

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