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Both models could explain our results.
Both models could explain why CXCL12-mediated cell motility was extinguished in CXCR7 knockdown cells at lower concentrations of CXCL12.
Both models could explain the TPS organization in cc11 isolates in which the tpsC1 cassette has apparently recombined into the tpsA locus.
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The models could explain 99% of variability in responses.
The results obtained from the new experimental conditions show that the predicted models could explain the process.
Nevertheless, even if the above models could explain two different times recorded for Er3+ emission, the short time observed for Er3+ seems to be much shorter than expected.
Outcomes of the spatial models could explain between 27%and70%0% of the variance of IMD, confirming the existence of a relationship between urban form and socio-economics across the six cities.
However, capacity-based models could explain the report of multiple sequential targets by referring to chunking mechanisms that would allow multiple stimuli to be part of one larger representation in memory.
Two formal models could explain how the regulation might work.
Variations in the experimental models could explain, at least in part, these differences.
At least two models could explain the RecA requirement for swarming motility.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com