The CD15 marker on the adenocarcinoma stainings indicated the disintegration of both inflammatory and epithelial cell membranes.
The expression of TGF-α mRNA and protein in both inflammatory and neoplastic cells indicates that TGF-α is directly synthesized by both cell compartments.
The receptor for advanced glycation endproducts [RAGE] is an induced receptor on both inflammatory and endothelial cells.
Thus we tested the expression of CCL5, a chemokine that targets both inflammatory and fibrogenic cells, and found a marked increase in comparison with WT mice on the same genetic background.
In this study, we used LN cells to interact with BMMSCs, where the LN population contains both inflammatory and regulatory T cell populations.
In addition, arthritic joints had higher levels of IFN-γ, and IL-4 than non-arthritic joints, suggesting that once target organ inflammation is initiated there is recruitment of both inflammatory and anti-inflammatory cell types.
MMPs are secreted by both inflammatory and connective tissue cells such as fibroblasts, fibroblast-related cells, chondrocytes, neutrophils, and monocytes/macrophages in response to both infectious assaults and inflammatory cytokines such as TNF-α and IL-1β [ 32, 33].
Additionally, the similar accumulation patterns of Wnt-responsive cells (siam +) and neutrophils/macrophages suggests that both inflammatory and Wnt signaling cells might respond to the same injury signals.
Finally, both inflammatory and vascular endothelial cells have been shown to express EPOR [53, 54].
Finally, both inflammatory and vascular endothelial cells have been shown to express EPOR [ 53, 54].
